Publications

Doepker CD, Heintz MM, van de Ligt JLG, Wikoff DS. 2021. Review of potential risks associated with supplementary dietary exposure to nitrate-containing compounds in swine — A paradox in light of emerging benefits. Trans Anim Sci 5(4):txab203; doi: 10.1093/tas/txab203. PMID: 34909600.

Chappell GA, Heintz MM, Borghoff SJ, Doepker CL, Wikoff DS. 2021. Lack of potential carcinogenicity for steviol glycosides — Systematic evaluation and integration of mechanistic data into the totality of evidence. Food Chem Toxicol 150(April):112045; doi: 10.1016/j.fct.2021.112045. PMID: 33587976.

Chappell GA, Heintz MM, Haws LC. 2021. Transcriptomic analyses of livers from mice exposed to 1,4-dioxane for up to 90 days to assess potential mode(s) of action underlying liver tumor development. Curr Res Toxicol 2:30–41; doi: 10.1016/j.crtox.2021.01.003. PMID: 34345848.

Heintz MM, McRee, R, Kumar, R, Baldwin, WS. 2020. Gender differences in diet-induced steatotic disease in Cyp2b-null mice. PLoS One 15(3):e0229896; doi: 10.1371/journal.pone.0229896. PMID: 32155178.

Heintz MM, Olack EM, Baldwin WS. Human CYP2B6 is an anti-obesity enzyme that produces active α-linolenic acid metabolites. Society of Toxicology 59th Annual Meeting, Virtual, March 2020.

Heintz MM, McRee R, Kumar R, Baldwin WS. Gender differences in diet-induced nonalcoholic steatohepatitis (NASH) in Cyp2b-null mice. Society of Toxicology 59th Annual Meeting, Virtual, March 2020.

Olack E, Heintz MM, Baldwin WS. Human CYP2B6 is an anti-obesity enzyme involved in unsaturated fatty acid metabolism. International Congress on Toxicology (ICTXV), Honolulu, HI, July 2019.

Heintz MM, Kumar R, Baldwin WS. Cyp2b-null male mice are susceptible to high-fat diet-induced obesity due to changes in PUFA metabolism and response to hepatic lipids as measured by RNAseq. International Congress on Toxicology (ICTXV), Honolulu, HI, July 2019.

Heintz MM, Kumar R, Rutledge M, Baldwin WS. 2019. Cyp2b-null male mice are susceptible to diet-induced obesity and perturbations in lipid homeostasis. J Nutr Biochem 70(August):125–137; doi: 10.1016/j.jnutbio.2019.05.004. PMID: 31202118.

Heintz MM, Sengupta N, Noorai R, Baldwin WS. Triclosan exposure represses development and alters sphingomyelin metabolism in Daphnia magna as determined by RNA-seq and lipidomics. 20^th Pollutant Responses in Marine Organisms, PRIMO20, Charleston, SC, May 2019.

Heintz MM, McRee R, Baldwin WS. The role of Cyp2b in diet-induced nonalcoholic steatohepatitis. Invited speaker: Clemson University Environmental Toxicology Program Seminar, Clemson, SC, April 2019.

Heintz MM, Kumar R, Baldwin WS. Cyp2b-null male mice are susceptible to high-fat diet-induced obesity due to changes in PUFA metabolism and response to hepatic lipids as measured by RNAseq. American College of Toxicology, 39th Annual Meeting, West Palm Beach, FL, November 2018.

Williams TL, Heintz MM, Baldwin WS. Several toxicants increase retention of triglycerides in Daphnia magna and human liver cells. Annual Biomedical Research Conference for Minority Students (ABRCMS), Indianapolis, IN, November 2018.

Williams TL, Heintz MM, Baldwin WS. Several toxicants increase retention of triglycerides in Daphnia magna and human liver cells. Research Experiences for Undergraduates (REUs), Alexandria, VA, October 2018.

Williams TL, Heintz MM, Baldwin WS. Several toxicants increase retention of triglycerides in Daphnia magna and human liver cells. Clemson Summer Research Symposium, July 2018.

Heintz MM, Kumar R, Baldwin WS. The role of Cyp2b in the metabolism of unsaturated fatty acids. Abstract #1291, Society of Toxicology 57th Annual Meeting, San Antonio, TX, March 2018.

McRee R, Heintz MM, Baldwin WS. Inhibition of CYP2B6 does not necessarily alter toxicity except in the case of chemicals with known active metabolites. Abstract #1292, Society of Toxicology 57th Annual Meeting, San Antonio, TX, March 2018.

Heintz MM, Brander SM, White JW. 2015. Endocrine disrupting compounds alter risk-taking behavior in guppies (Poecilia reticulata). Ethology 121(5):480–491; doi: 10.1111/eth.12362.